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In addition, fatty acid metabolic reprogramming in GA drives distinct pathways: β-oxidation in hepatocytes and macrophages promotes urate synthesis and proinflammatory cytokine release (18, 64, 93), AA metabolism in neutrophils drives the LTB4-BLT1-mediated chemotaxis and NETosis, amplifying local inflammation (76–78)

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This contributes to both its healing properties (maintaining blood supply to injured tissue) and its protective effects against various forms of organ damage