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TB-500 also exhibits clinically relevant anti-inflammatory properties through downregulation of inflammatory cytokines including TNF-α and IL-1β

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PON1 is susceptible to being partially inactivated by the metabolic products of oxidative stress—specifically, by oxidized glutathione (GSSG) through the process of S-glutathionylation and by products of lipid peroxidation, which can also react with the free sulfhydryl group of the enzyme (Aviram et al., 1999