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( + T cells through buccal mucosa cell EWAS (e.g., TNFAIP3, IRF5), demonstrating that epigenetic features of non-invasive samples (such as buccal cells) can be used for early diagnosis and risk assessment of RA ( IL6ST , SOCS3 ) significantly associated with drug responsiveness, providing a predictive tool for personalised treatment ( The core role of epigenetic regulation in the common mechanisms of ADs has been further revealed: the majority of DMRs specific to regulatory T cells (Tregs) are contained within Treg-specific super-enhancers and are closely related to the transcription and other epigenetic changes of both naïve and effector Tregs ( Current epigenomics research has driven innovations in the management of ADs from three dimensions: the development of diagnostic biomarkers, in-depth mechanistic dissection, and optimisation of therapeutic strategies
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Unknowns include high-dose impacts, long-term child health outcomes, and possible unexpected reactions
