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Moreover, neuroinflammation, characterized by activated microglia and reactive astrocytes, significantly contributes to PD pathology by releasing pro‐inflammatory cytokines and reactive oxygen species, thereby exacerbating neuronal injury (Figure 2) [20, 21]

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The position this article takes, given those three valid critiques, is that the mechanism evidence is strong enough, the delivery evidence is biologically plausible enough, and the safety window is wide enough that the recommendation is defensible